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Meanwhile, the diagnosis of the disease is the same for various clinical variants of hemochromatosis. After assessing the clinical signs, patients are assigned a set of laboratory and instrumental studies to verify the legitimacy of azithromycin drug.
Laboratory criteria for hemochromatosis are a significant increase in the level of iron, ferritin and transferrin in the blood serum, an increase in the excretion of iron in the urine, and a decrease in the total iron-binding capacity of the blood serum. Diagnosis is confirmed by needle biopsy of the liver or skin, which shows hemosiderin deposits. The hereditary nature of hemochromatosis is established as a result of generic drug genetic diagnosis.
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In order to assess the severity of damage to internal organs and the prognosis of the disease, liver tests, blood and urine glucose levels, glycosylated hemoglobin, etc. are examined. Laboratory diagnosis of hemochromatosis is supplemented by instrumental studies. Buy zithromax 500mg of the joints, ECG, echocardiography, ultrasound of the abdominal organs, MRI of the liver, etc.
The main goal of the therapy is to remove excess iron from the body and prevent the development of complications. Patients with hemochromatosis are prescribed a diet that restricts foods high in iron (apples, meat, liver, buckwheat, spinach, etc.), easily digestible carbohydrates. It is forbidden to take multivitamins, ascorbic acid, dietary supplements containing iron, alcohol.
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To remove excess iron from the body, they resort to bloodletting under the control of hemoglobin, blood hematocrit, and ferritin. For the same purpose, extracorporeal methods of hemocorrection can be used - plasmapheresis, hemosorption, cytapheresis.
Pathogenetic drug therapy for hemochromatosis is based on intramuscular or intravenous administration of deferoxamine, which binds Fe3+ ions, to the patient. At the same time, symptomatic treatment of liver cirrhosis, heart failure, diabetes mellitus, hypogonadism is carried out. With severe arthropathy, indications for arthroplasty (arthroplasty of the affected joints) are determined.
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Despite the progressive course of the disease, timely therapy can prolong the life of patients with hemochromatosis for several decades. In the absence of treatment, the average life expectancy of patients after the diagnosis of pathology does not exceed 4-5 years. The presence of complications of hemochromatosis (mainly liver cirrhosis and congestive heart failure) is a poor prognostic sign.
In hereditary hemochromatosis, prevention is reduced to buy oral azithromycin 500mg screening, early detection and initiation of treatment of the disease. To avoid the development of secondary hemochromatosis allows rational nutrition, control over the appointment and intake of iron preparations, blood transfusions, refusal to take alcohol, monitoring patients with diseases of the liver and blood system.
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The history of hemochromatosis as a disease (a symptom complex, a condition characterized by excessive accumulation of iron (Fe) in the body) originates from the end of the 19th century, namely from 1871, but the current name for the pathology stuck only 18 years later (1889) . Hemachromatosis (HC) is also called pigmentary cirrhosis and bronze diabetes, which, in principle, reflects its clinical manifestations. discoloration of the skin (to bronze), all signs of diabetes mellitus and degeneration of the hepatic parenchyma with the development of cirrhosis.
In addition, hemochromatosis is called generalized hemosiderosis, von Recklinghausen-Appelbaum disease and Troisier-Anot-Choffard syndrome. Formation of this symptomThe mocomplex ultimately leads to damage to many organs and to the development of azithromycin drug organ failure.